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Maternal inflammation during pregnancy triggered autism-spectrum behaviors in offspring mice, including excessive brain activity, heightened sensory responses, and repetitive actions alongside elevated seizure susceptibility. The condition persisted into adulthood, creating what researchers characterized as a stable neurological state resistant to typical interventions.
A single administration of rapamycin produced dramatic improvements across nearly all measured symptoms within approximately two hours of treatment. While the therapeutic effects proved temporary rather than permanent, the rapid response raised significant questions about the functional capacity of mature neural circuits.
The findings challenge longstanding neuroscience assumptions regarding adult brain plasticity and adaptability. Researchers suggest that even established brain dysfunction may retain greater potential for modification than previously recognized, opening possibilities for novel therapeutic approaches to neurodevelopmental conditions.
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